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for postoperative morbidity was used (grade I–V)

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mechanism is plausible and supported by subsequent work, but human clinical confirmation is still emerging.) A 2023 Science paper extended this finding to the gut microbiome broadly, showing that microbial DPP-4 can disrupt glucose metabolism in mice with impaired gut barrier function — and that current human DPP-4 inhibitors (gliptins) have little effect on microbial DPP-4 isoforms (10)

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